Vitamin E is beneficial for the absorption of vitamin A and carotene as well as storage in the liver. Experiments have shown that vitamin E protects vitamin A and carotene from oxidation in the intestines. In recent years, it has been suggested that for chickens, there is an antagonistic effect between vitamin E and A, that is, high levels of vitamin A in the diet can lower the level of vitamin E in plasma and body fat.
Vitamin E promotes the conversion of carotene to vitamin A. Insufficient vitamin E can also affect the synthesis of vitamin C in the body. Vitamin C can alleviate the symptoms caused by vitamins A, E, thiamine, riboflavin, vitamin B12 and pantothenic acid. Folic acid can promote the synthesis of vitamin C in the intestinal microbes of animals.
Vitamin A in rats promotes the synthesis of vitamin C. In the absence of thiamine in rats, it affects the utilization of riboflavin and increases its urinary output. In the absence of riboflavin, the amount of thiamine in the body tissue decreases, but does not affect the amount of urine. In the absence of riboflavin, the formation of niacin by tryptophan is blocked and niacin deficiency occurs.
Vitamin B12 can increase the utilization of folic acid and promote the synthesis of choline. When vitamin B12 is insufficient in the diet of the breeder, the amount of pantothenic acid is increased. When pantothenic acid is insufficient, vitamin B12 deficiency can be aggravated. Insufficient vitamin B6 affects the absorption of vitamin B12 and increases the amount of B12 excreted in the feces. In addition, there is an interaction between biotin and vitamin C and other vitamins between vitamins.
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